Vasconcelos, ATR, Ferreira HB, Bizarro CV, Bonatto SL, Carvalho MO, Pinto PM, Almeida DF, Almeida LGP, Almeida R, Alves-Filho L, Assunção EN, Azevedo VAC, Bogo MR, Brigido MM, Brocchi M, Burity HA, Camargo AA, Camargo SS, Carepo MS, Carraro DM, De Mattos Cascardo JC, Castro LA, Cavalcanti G, Chemale G, Collevatti RG, Cunha CW, Dallagiovanna B, Dambrós BP, Dellagostin OA, Falcão C, Fantinatti-Garboggini F, Felipe MSS, Fiorentin L, Franco GR, Freitas NSA, Frías D, Grangeiro TB, Grisard EC, Guimarães CT, Hungria M, Jardim SN, Krieger MA, Laurino JP, Lima LFA, Lopes MI, Loreto ÉLS, Madeira HMF, Manfio GP, Maranhão AQ, Martinkovics CT, Medeiros SRB, Moreira MAM, Neiva M, Ramalho-Neto CE, Nicolás MF, Oliveira SC, Paixão RFC, Pedrosa FO, Pena SDJ, Pereira M, Pereira-Ferrari L, Piffer I, Pinto LS, Potrich DP, Salim ACM, Santos FR, Schmitt R, Schneider MPC, Schrank A, Schrank IS, Schuck AF, Seuanez HN, Silva DW, Silva R, Silva SC, Soares CMA, Souza KRL, Souza RC, Staats CC, Steffens MBR, Teixeira SMR, Urmenyi TP, Vainstein MH, Zuccherato LW, Simpson AJG, Zaha A.
2005.
Swine and poultry pathogens: The complete genome sequences of two strains of Mycoplasma hyopneumoniae and a strain of Mycoplasma synoviae. Journal of Bacteriology. 187:5568–5577., Number 16
Abstractn/a
Gonçalves, TL, Erthal F, Corte CLD, Müller LG, Piovezan CM, Nogueira CW, Rocha JBT.
2005.
{Involvement of oxidative stress in the pre-malignant and malignant states of cervical cancer in women}. Clinical Biochemistry. 38, Number 12
AbstractObjectives: To evaluate the potential role of oxidative stress in the evolution of cervical cancer, including its pre-malignant states. Design and methods: Erythrocytes thiobarbituric acid reactive substances (TBARS) levels, plasma vitamin C and thiol content and total blood $δ$-ALA-D levels were estimated in 46 untreated cervical cancer and pre-malignant patients and in 46 age-sex-matched controls. Results: Erythrocytes from patients, regardless of disease state, pre-malignant (low squamous intraepithelial lesion-LSIL and high squamous intraepithelial lesion-HSIL) or cancer, showed a significant 2-3 times increase in TBARS levels (P {\textless} 0.01). Plasma vitamin C was lower in the carcinoma group (P {\textless} 0.01). The reactivation index of $δ$-aminolevulinate dehydratase ($δ$-ALA-D) was higher in the patient group, when compared to control (P {\textless} 0.01). Conclusion: LSIL, HSIL or cervical cancer can be associated with changes in 3 indicators of oxidative stress: increase in erythrocyte TBARS, ALA-D reactivation index and a decrease in vitamin C content, that may play an important role in carcinogenesis. © 2005 The Canadian Society of Clinical Chemists. All rights reserved.
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